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4-Hydroxynonenal Antibodies

We offer 4-Hydroxynonenal Antibodies for use in common research applications: ELISA, Simple Western, Western Blot. Each 4-Hydroxynonenal Antibody is fully covered by our Guarantee+, to give you complete peace of mind and the support when you need it. Our 4-Hydroxynonenal Antibodies can be used in a variety of model species: Multi-Species. Use the list below to choose the 4-Hydroxynonenal Antibody which is most appropriate for your research; you can click on each one to view full technical details, images, references, reviews and related products. Choose from our 4-Hydroxynonenal polyclonal antibodies and browse our 4-Hydroxynonenal monoclonal antibody catalog.

Alternate Names for 4-Hydroxynonenal Antibodies

anti-4-Hydroxynonenal antibody, anti-4 HNE antibody

2 Results for "4-hydroxynonenal" in Primary Antibodies

MAB3249

4-Hydroxynonenal Antibody (198960) [Unconjugated]

(1 Review)

(117 Publications)

100 μg

MAB3249

Western blot shows lysates of HepG2 human hepatocellular carcinoma cell line untreated (-) or treated (+) with 4-Hydroxynonenal (4-HNE). PVDF membrane was probed with 1 µg/mL of Mouse Anti-4-Hydroxynonenal Monoclonal Antibody (Catalog # MAB3249) followed by HRP-conjugated Anti-Mouse IgG Secondary Antibody (Catalog # HAF007). Specific bands were detected for 4-Hydroxynonenal at adducts of histidine residues (as indicated). This experiment was conducted under reducing conditions and using Immunoblot Buffer Group 1.

<b>Biological Strategies Validation. </b>Simple Western lane view shows lysates of HepG2 human hepatocellular carcinoma cell line untreated (-) or treated (+) with 4-Hydroxynonenal (4-HNE), loaded at 0.2 mg/mL. Specific bands were detected for 4‑Hydroxynonenal at adducts of histidine residues kDa (as indicated) using 5 µg/mL of Mouse Anti-4‑Hydroxynonenal Monoclonal Antibody (Catalog # MAB3249). This experiment was conducted under reducing conditions and using the 12-230 kDa separation system.

4‑Hydroxynonenal was detected in immersion fixed paraffin-embedded sections of human prostate using Mouse Anti-4‑Hydroxynonenal Monoclonal Antibody (Catalog # MAB3249) at 0.1 µg/mL for 1 hour at room temperature followed by incubation with the Anti-Mouse IgG VisUCyte™ HRP Polymer Antibody (Catalog # VC001). Before incubation with the primary antibody, tissue was subjected to heat-induced epitope retrieval using Antigen Retrieval Reagent-Basic (Catalog # CTS013). Tissue was stained using DAB (brown) and counterstained with hematoxylin (blue). Specific staining was localized to cytoplasm. View our protocol for IHC Staining with VisUCyte HRP Polymer Detection Reagents.

4‑Hydroxynonenal was detected in immersion fixed paraffin-embedded sections of human prostate cancer tissue using Mouse Anti-4‑Hydroxynonenal Monoclonal Antibody (Catalog # MAB3249) at 0.5 µg/mL for 1 hour at room temperature followed by incubation with the Anti-Mouse IgG VisUCyte™ HRP Polymer Antibody (Catalog # VC001). Before incubation with the primary antibody, tissue was subjected to heat-induced epitope retrieval using Antigen Retrieval Reagent-Basic (Catalog # CTS013). Tissue was stained using DAB (brown) and counterstained with hematoxylin (blue). Specific staining was localized to cytoplasm. View our protocol for IHC Staining with VisUCyte HRP Polymer Detection Reagents.

Diapocynin inhibits the formation of 3-nitrotyrosine (3-NT) and 4-hydroxynonenal (4-HNE) in the substantia nigra (SN) of MPTP-treated mice. (A) Representative Western blots illustrating the expression of 3-NT in SN. (B) Bar graph showing mean Western blot 3-NT/ beta -actin ratios ± SEM in SN of 6 mice per group. (C) Double labeling of tyrosine hydroxylase (TH) and 3-NT in SN region of ventral midbrain. (D) Representative Western blots illustrating the expression of 4-HNE in SN. (E) Bar graph showing mean Western blot 4-HNE/ beta -actin ratios ± SEM in SN of 6 mice per group. (F) Double labeling of TH and 4-HNE in SN region of ventral midbrain. Images were captured at 60× magnification. The SN zone is outlined in white dots. Inset pictures demonstrated colocalization of TH and 3-NT/4-HNE. ***P <0.001 vs the control group; **P <0.01 vs the control group; *P <0.05 vs the MPTP group; #P <0.001 vs the MPTP group. Image collected and cropped by CiteAb from the following publication (//pubmed.ncbi.nlm.nih.gov/23092448), licensed under a CC-BY license. Not internally tested by R&D Systems.

Diapocynin inhibits the formation of 3-nitrotyrosine (3-NT) and 4-hydroxynonenal (4-HNE) in the substantia nigra (SN) of MPTP-treated mice. (A) Representative Western blots illustrating the expression of 3-NT in SN. (B) Bar graph showing mean Western blot 3-NT/ beta -actin ratios ± SEM in SN of 6 mice per group. (C) Double labeling of tyrosine hydroxylase (TH) and 3-NT in SN region of ventral midbrain. (D) Representative Western blots illustrating the expression of 4-HNE in SN. (E) Bar graph showing mean Western blot 4-HNE/ beta -actin ratios ± SEM in SN of 6 mice per group. (F) Double labeling of TH and 4-HNE in SN region of ventral midbrain. Images were captured at 60× magnification. The SN zone is outlined in white dots. Inset pictures demonstrated colocalization of TH and 3-NT/4-HNE. ***P <0.001 vs the control group; **P <0.01 vs the control group; *P <0.05 vs the MPTP group; #P <0.001 vs the MPTP group. Image collected and cropped by CiteAb from the following publication (//pubmed.ncbi.nlm.nih.gov/23092448), licensed under a CC-BY license. Not internally tested by R&D Systems.

Analysis of inflammatory response and oxidative stress levels of atherosclerotic VSMCs in mice: (A–E) representative images (A) and data of the protein levels of 4-HNE (B), Nrf2 (C), TNF-alpha (D), and IL-1 beta (E). (F–H) The expression of Nrf2, TNF-alpha , and IL-1 beta was quantified by qPCR and standardized to GAPDH. (I,O) ROS fluorescence detection (red) and quantification. Scale bar 100 μm. (J–N) Representative images (J) and data of the protein levels of P-PI3K (K), PI3K (L), P-Akt (M), and Akt (N). The bar graphs depict means ± SEM; statistical significance * p < 0.05, ** p < 0.01, and *** p < 0.001 and ns by the one-way ANOVA test. Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/37047626), licensed under a CC-BY license. Not internally tested by R&D Systems.

Analysis of inflammatory response and oxidative stress levels of atherosclerotic VSMCs in mice: (A–E) representative images (A) and data of the protein levels of 4-HNE (B), Nrf2 (C), TNF-alpha (D), and IL-1 beta (E). (F–H) The expression of Nrf2, TNF-alpha , and IL-1 beta was quantified by qPCR and standardized to GAPDH. (I,O) ROS fluorescence detection (red) and quantification. Scale bar 100 μm. (J–N) Representative images (J) and data of the protein levels of P-PI3K (K), PI3K (L), P-Akt (M), and Akt (N). The bar graphs depict means ± SEM; statistical significance * p < 0.05, ** p < 0.01, and *** p < 0.001 and ns by the one-way ANOVA test. Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/37047626), licensed under a CC-BY license. Not internally tested by R&D Systems.

CYP1B1 expression attenuates the effect of anti-PD-1 therapy. A Western blot analysis of MC38 cells expressing ctrl shRNA or CYP1B1 shRNAs. B C57BL/6 J mice carrying the indicated MC38 cell formed tumors at the right flank were treated with anti-mPD-1 antibody or isotype control IgG for a total of five treatments. C, D Tumor images C and tumor growth curve D in mice bearing indicated MC38 cell tumors treated with anti-mPD-1 or isotype control IgG antibodies. E IHC staining of 4-HNE in MC38 tumors expressing ctrl or CYP1B1 shRNA. F Quantification of 4-HNE IHC staining. n = 5 per group. p value was determined by Student’s two-sided t test. *p < 0.05, **p < 0.01, ***p < 0.001. Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/37059712), licensed under a CC-BY license. Not internally tested by R&D Systems.

B cell-derived antibodies mediate HHcy-aggravated kidney lipid peroxidation in hypertensive mice. a Immunohistochemical staining of 4-HNE (brown) from kidney sections in sham or 2K1C mice with or without Hcy (1.8 g/L) in drinking water for 4 weeks, indicative of lipid peroxidation. Rituximab was administered to HHcy 2K1C mice at the start of modeling (i.p. 75 μg/20 g body weights every other day for 4 weeks). The immunohistochemical staining was calculated as the percentage of brown signals over the total area. n = 3. b, c Effects of 2K1C and HHcy on lipid peroxidation in the kidney were measured using ELISA for LPO (b) and MDA (c). n = 6. d Quantitative PCR analysis of redox enzyme expression in kidney tissues, including acsl4, lox15, slc7a11 and gpx4. n = 6. e Western blot analysis of LOX15, GPX4 and SLC7A11 protein expression and quantification. beta -actin was used as an internal control. n = 3. f, g The redox balance in the kidney was assayed for the ratio of GSSG/GSH and NADP+/NADPH using ELISA. n = 6. h Quantitative PCR analysis of redox enzyme expression in the renal CD31-positive GECs from frozen section by laser capture microdissection, including acsl4, lox15, slc7a11 and gpx4. n = 3. LPO lipid peroxide, MDA malondialdehyde, 4-HNE 4-hydroxynonenal, lox15 lipoxygenase 15, acsl4 acyl-CoA synthetase long chain family member 4, slc7a11 cystine/glutamate transporter, gpx4 glutathione peroxidase 4, GSH glutathione, GSSG glutathione disulfide. All data are expressed as the means ± SEM. *P < 0.05, **P < 0.01 Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/36907919), licensed under a CC-BY license. Not internally tested by R&D Systems.

CYP1B1 expression attenuates the effect of anti-PD-1 therapy. A Western blot analysis of MC38 cells expressing ctrl shRNA or CYP1B1 shRNAs. B C57BL/6 J mice carrying the indicated MC38 cell formed tumors at the right flank were treated with anti-mPD-1 antibody or isotype control IgG for a total of five treatments. C, D Tumor images C and tumor growth curve D in mice bearing indicated MC38 cell tumors treated with anti-mPD-1 or isotype control IgG antibodies. E IHC staining of 4-HNE in MC38 tumors expressing ctrl or CYP1B1 shRNA. F Quantification of 4-HNE IHC staining. n = 5 per group. p value was determined by Student’s two-sided t test. *p < 0.05, **p < 0.01, ***p < 0.001. Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/37059712), licensed under a CC-BY license. Not internally tested by R&D Systems.

B cell-derived antibodies mediate HHcy-aggravated kidney lipid peroxidation in hypertensive mice. a Immunohistochemical staining of 4-HNE (brown) from kidney sections in sham or 2K1C mice with or without Hcy (1.8 g/L) in drinking water for 4 weeks, indicative of lipid peroxidation. Rituximab was administered to HHcy 2K1C mice at the start of modeling (i.p. 75 μg/20 g body weights every other day for 4 weeks). The immunohistochemical staining was calculated as the percentage of brown signals over the total area. n = 3. b, c Effects of 2K1C and HHcy on lipid peroxidation in the kidney were measured using ELISA for LPO (b) and MDA (c). n = 6. d Quantitative PCR analysis of redox enzyme expression in kidney tissues, including acsl4, lox15, slc7a11 and gpx4. n = 6. e Western blot analysis of LOX15, GPX4 and SLC7A11 protein expression and quantification. beta -actin was used as an internal control. n = 3. f, g The redox balance in the kidney was assayed for the ratio of GSSG/GSH and NADP+/NADPH using ELISA. n = 6. h Quantitative PCR analysis of redox enzyme expression in the renal CD31-positive GECs from frozen section by laser capture microdissection, including acsl4, lox15, slc7a11 and gpx4. n = 3. LPO lipid peroxide, MDA malondialdehyde, 4-HNE 4-hydroxynonenal, lox15 lipoxygenase 15, acsl4 acyl-CoA synthetase long chain family member 4, slc7a11 cystine/glutamate transporter, gpx4 glutathione peroxidase 4, GSH glutathione, GSSG glutathione disulfide. All data are expressed as the means ± SEM. *P < 0.05, **P < 0.01 Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/36907919), licensed under a CC-BY license. Not internally tested by R&D Systems.

RMZL suppressed oxidative stress and inflammation in HIRI mice. Mice were subjected to ischemia for 1 h and then reperfusion for 6 h through liver surgery, and followed by RMZL treatment. (A) SOD and MDA levels in the left hepatic tissues were determined using commercial kits. (C) 4-HNE expression in the left hepatic tissues was detected by IHC. (D) The levels of TNF-alpha , IL-1 beta , IL-6 and IL-10 in serum from mice were evaluated using ELISA. All data were indicated as mean ± SD (n = 6 per group). *P < 0.05, **P < 0.01, ***P < 0.001 Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/40263992), licensed under a CC-BY license. Not internally tested by R&D Systems.

S. aureus and E. coli infections induced lipid peroxides accumulation in infected bone microenvironment and BMSCs. A) Lipid peroxidation levels in bone marrow on Day 3 post‐infection in murine osteomyelitis model induced by S. aureus and E. coli were determined by WB analysis of 4‐HNE and MDA protein modifications. Each group contained four mice. B) Representative tissue immunofluorescence images of LepR, MDA, 4‐HNE, ACSL4, GPX4 in the uninfected murine femur and implant‐associated bone infection model induced by S. aureus and E. coli. Scale bar = 50 µm. C) The schematic route of animal experiments and gating strategy were depicted. D) Lipid peroxide regulation by S. aureus and E. coli infections and Fer‐1 in CD45−CD31−Ter119−LepR+ BMSCs were analyzed in murine osteomyelitis model by Liperfluo staining and analyzed by flow cytometry. E,F) Quantification of median fluorescent intensity in flow cytometry analysis in (D). Values are means ± SDs. Each group contained six mice. Multiple comparison was performed by one‐way analysis of variance (ANOVA) with Tukey's post‐hoc analysis. Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/39166412), licensed under a CC-BY license. Not internally tested by R&D Systems.

Inhibition of H3K14la improved functional outcomes after ICH in mice. Immunostaining of H3K14la and NeuN was performed at 24 h after ICH. “H” indicates hematoma. The representative images (A) and quantifications (B) are shown. n = 5 mice. C The expression of 4-HNE was measured at 24 h after ICH by western blot analysis. Tubulin serves as the loading control. n = 4 mice. D FJC staining was performed at 24 h after ICH. The representative images and quantification are shown. n = 4 mice. E–G 1 μl of 500 μM GSK or 10 mM Caloxin 2A1 was injected at 30 min after ICH. Neurological function and motor function were determined at indicated time points after ICH. n = 8 mice. FJC staining was performed at 24 h after ICH. The representative images (H) and quantification (I) are shown. n = 4 mice. J–L AAV virus of NC or shP300 virus was injected at 1 week before ICH and Neurological function and motor function were examined at indicated time points after ICH. Results are shown as scatter plots (Mean ± SD). n = 8 mice. One-way (B–D and I) or two-way (E–G and J–L) ANOVA followed by Tukey’s multiple comparisons tests was performed, *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001vs Sham; #p < 0.05 vs ICH + NC/Veh; NS, not significant. Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/40701963), licensed under a CC-BY license. Not internally tested by R&D Systems.

RMZL suppressed oxidative stress and inflammation in HIRI mice. Mice were subjected to ischemia for 1 h and then reperfusion for 6 h through liver surgery, and followed by RMZL treatment. (A) SOD and MDA levels in the left hepatic tissues were determined using commercial kits. (C) 4-HNE expression in the left hepatic tissues was detected by IHC. (D) The levels of TNF-alpha , IL-1 beta , IL-6 and IL-10 in serum from mice were evaluated using ELISA. All data were indicated as mean ± SD (n = 6 per group). *P < 0.05, **P < 0.01, ***P < 0.001 Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/40263992), licensed under a CC-BY license. Not internally tested by R&D Systems.

Reactivity:

All-Multi

Species Glossary

Reactivity: All-Multi

Applications:

, ,

Applications: , ,

Host:

Mouse Monoclonal

Host: Mouse Monoclonal

Sizes:

Sample Size Available

Sizes: Sample Size Available

Conjugate:

Unconjugated

Available Conjugates:

Validated by:

Validated by:

Conjugate	Catalog #	Availability	Size	Price
Alexa Fluor 350	FAB3249U-100UG
Alexa Fluor 405	FAB3249V-100UG
Alexa Fluor 488	FAB3249G-100UG
Alexa Fluor 532	FAB3249X-100UG
Alexa Fluor 594	FAB3249T-100UG
Alexa Fluor 647	FAB3249R-100UG
Alexa Fluor 700	FAB3249N-100UG
Alexa Fluor 750	FAB3249S-100UG
Allophycocyanin	FAB3249A
Biotin	FAB3249B
CoraFluor 1	FAB3249CL1
DyLight 350	FAB3249D
DyLight 405	FAB3249E
DyLight 488	FAB3249K
DyLight 550	FAB3249L
DyLight 594	FAB3249M
DyLight 650	FAB3249W
DyLight 680	FAB3249Y
DyLight 755	FAB3249Z
FITC	FAB3249F
HRP	FAB3249H
Janelia Fluor 525	FAB3249JF525
Janelia Fluor 549	FAB3249I
Janelia Fluor 646	FAB3249J
Janelia Fluor 669	FAB3249JF669
PE	FAB3249P
PerCP	FAB3249C
mFluor Violet 450 SE	FAB3249MFV450
mFluor Violet 500 SE	FAB3249MFV500
mFluor Violet 610 SE	FAB3249MFV610

Available Formulations

Formulation	Catalog #	Availability	Price
Carrier Free	MAB3249-SP

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NBP2-59353

4-Hydroxynonenal Antibody (12F7) - BSA Free

(3 Publications)

100 μg

NBP2-59353

Western Blot: 4-Hydroxynonenal Antibody (12F7) [NBP2-59353] - Western Blot analysis of 4-hydroxy-nonenal-BSA Conjugate showing detection of 67 kDa 4-hydroxy-nonenal protein using Mouse Anti-4-hydroxy-nonenal Monoclonal Antibody, Clone 12F7 (NBP2-59353). Lane 1: Molecular Weight Ladder (MW). Lane 2: BSA (0.5 ug). Lane 3: 4-hydroxyl nonenal-BSA (0.5 ug). Lane 4: 4-hydroxy nonenal-BSA (2.0 ug). Lane 5: 4-hydroxy-2-hexenal (0.5 ug). Lane 6: 4-hydroxy-2-hexenal (2.0 ug). Block: 5% Skim Milk in TBST. Primary Antibody: Mouse Anti-4-hydroxy-nonenal Monoclonal Antibody (NBP2-59353) at 1:1000 for 2 hours at RT. Secondary Antibody: Goat Anti-Mouse IgG: HRP at 1:2000 for 60 min at RT. Color Development: ECL solution for 5 min in RT. Predicted/Observed Size: 67 kDa.

Immunocytochemistry/Immunofluorescence: 4-Hydroxynonenal Antibody (12F7) [NBP2-59353] - Immunocytochemistry/Immunofluorescence analysis using Mouse Anti-4-Hydroxynonenal Monoclonal Antibody, Clone 12F7 (NBP2-59353). Tissue: Embryonic kidney epithelial cell line (HEK293). Species: Human. Fixation: 5% Formaldehyde for 5 min. Primary Antibody: Mouse Anti-4-Hydroxynonenal Monoclonal Antibody (NBP2-59353) at 1:50 for 30-60 min at RT. Secondary Antibody: Goat Anti-Mouse Alexa Fluor 488 at 1:1500 for 30-60 min at RT. Counterstain: Phalloidin Alexa Fluor 633 F-Actin stain; DAPI (blue) nuclear stain at 1:250, 1:50000 for 30-60 min at RT. Magnification: 20X (2X Zoom). (A,C,E,G) - Untreated. (B,D,F,H) - Cells cultured overnight with 50 M H2O2. (A,B) DAPI (blue) nuclear stain. (C,D) Phalloidin Alexa Fluor 633 F-Actin stain. (E,F) 4-Hydroxynonenal Antibody. (G,H) Composite. Courtesy of: Dr. Robert Burke, University of Victoria.

Immunocytochemistry/Immunofluorescence: 4-Hydroxynonenal Antibody (12F7) [NBP2-59353] - Tissue: Embryonic kidney cells (HEK293). Species: Human. Fixation: 5% Formaldehyde for 5 min. Primary Antibody: Mouse Anti-4-Hydroxynonenal Monoclonal Antibody at 1:50 for 30-60 min at RT. Secondary Antibody: Goat Anti-Mouse Alexa Fluor 488 at 1:1500 for 30-60 min at RT. Counterstain: Phalloidin Alexa Fluor 633 F-Actin stain; DAPI (blue) nuclear stain at 1:250, 1:50000 for 30-60 min at RT. Magnification: 20X (2X Zoom). Courtesy of: Dr. Robert Burke, University of Victoria.

Decreased SLC7A11 expression augments the susceptibility of CARD9-deficient MDSCs to ferroptosis during C. tropicalis stimulation. (A and B) WT and Card9−/− MDSCs were stimulated with C. tropicalis (MOI = 1) for 6 h. The expression of Slc7a11, Slc3a2 and Gpx4 mRNA were detected by qPCR. (C and D) WT and Card9−/− MDSCs were stimulated with C. tropicalis (MOI = 1, 2, 4) for 24 h. The protein levels of SLC7A11 and GPX4 were measured by western blot. (E-G) WT and Card9−/− MDSCs were stimulated with C. tropicalis (MOI = 1) for 24 h. The glutamate in medium supernatant, intracellular cystine and GSH were determined by the corresponding assay kit. (H-L) WT and Card9−/− MDSCs were stimulated with C. tropicalis (MOI = 1), meanwhile, only CARD9-deficient MDSCs were treated with vehicle (DMSO) or ferrostatin-1 (Fer-1, 10 uM). After 24 h, the proportion of dead MDSCs was measured by 7AAD (H). The relative cell viability of MDSCs was detected by CCK-8 kit (I). The Lipid ROS of MDSCs was measured by BODIPY 581/591 C11 staining (J). The protein level of 4HNE was determined by western blot (K). The relative MDA level in MDSCs was detected by MDA Assay Kit (L). Data with error bars are expressed as mean +/- SEM, n = 3. Each panel shows at least three independent biological replicates from a typical experiment. Statistical analysis was determined by one-way or two-way ANOVA with Bonferroni’s multiple comparisons test for post-hoc test. ns (not significant), P > 0.05; *P < 0.05, **P < 0.01, ***P < 0.001 Image collected and cropped by CiteAb from the following open publication (//pubmed.ncbi.nlm.nih.gov/38566195), licensed under a CC-BY license. Not internally tested by Novus Biologicals.

Reactivity:

All-NA

Species Glossary

Reactivity: All-NA

Applications:

, ,

Applications: , ,

Host:

Mouse Monoclonal

Host: Mouse Monoclonal

Sizes:

Sample Size Available

Sizes: Sample Size Available

Conjugate:

Unconjugated

Available Conjugates:

Conjugate	Catalog #	Availability	Size	Price
Alexa Fluor 350	NBP2-59353AF350
Alexa Fluor 405	NBP2-59353AF405
Alexa Fluor 488	NBP2-59353AF488
Alexa Fluor 532	NBP2-59353AF532
Alexa Fluor 594	NBP2-59353AF594
Alexa Fluor 647	NBP2-59353AF647
Alexa Fluor 700	NBP2-59353AF700
Alexa Fluor 750	NBP2-59353AF750
Alexa Fluor Plus 405	NBP2-59353AFP405
Alexa Fluor Plus 488	NBP2-59353AFP488
Alexa Fluor Plus 555	NBP2-59353AFP555
Alexa Fluor Plus 594	NBP2-59353AFP594
Alexa Fluor Plus 647	NBP2-59353AFP647
Alexa Fluor Plus 680	NBP2-59353AFP680
Allophycocyanin	NBP2-59353APC
Biotin	NBP2-59353B
CoraFluor 1	NBP2-59353CL1
DyLight 350	NBP2-59353UV
DyLight 405	NBP2-59353V
DyLight 488	NBP2-59353G
DyLight 550	NBP2-59353R
DyLight 594	NBP2-59353DL594
DyLight 650	NBP2-59353C
DyLight 680	NBP2-59353FR
DyLight 755	NBP2-59353IR
FITC	NBP2-59353F
HRP	NBP2-59353H
Janelia Fluor 525	NBP2-59353JF525
Janelia Fluor 549	NBP2-59353JF549
Janelia Fluor 646	NBP2-59353JF646
Janelia Fluor 669	NBP2-59353JF669
PE	NBP2-59353PE
PerCP	NBP2-59353PCP
mFluor Violet 450 SE	NBP2-59353MFV450
mFluor Violet 500 SE	NBP2-59353MFV500
mFluor Violet 610 SE	NBP2-59353MFV610

Available Formulations

Formulation	Catalog #	Availability	Price
BSA Free	NBP2-59353-25ug

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