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AB-108-C, open histogram), followed by Allophycocyanin-conjugated Anti-Goat IgG Secondary Antibody (Catalog # F0108). To facilitate intracellular staining, cells were fixed with Flow Cytometry Fixation Buffer (Catalog # FC004) and permeabilized with Flow Cytometry Permeabilization/Wash Buffer I (Catalog # FC005). View our protocol for Staining Intracellular Molecules." class="big_lightbox" target="_blank">
Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD). LORD is an autosomal dominant disorder characterized by onset in the fifth to sixth decade with night blindness and punctate yellow-white deposits in the retinal fundus, progressing to severe central and peripheral degeneration, with choroidal neovascularization and chorioretinal atrophy.; Subcellular location: Secreted
![Immunohistochemistry C1qTNF5/CTRP5 Antibody (332923) [Unconjugated]](http://images.novusbio.com/fullsize/antibody/C1qTNF5_MAB3167_Immunohistochemistry_19941.jpg)
![Intracellular Staining by Flow Cytometry C1qTNF5/CTRP5 Antibody [Unconjugated]](http://images.novusbio.com/images/antibody/C1qTNF5_AF3167_Flow_Cytometry_19755.jpg)
![Bioactivity C1qTNF5/CTRP5 [Unconjugated]](http://images.novusbio.com/fullsize/protein/CTRP5_9510TN_1624.jpg)
![Intracellular Staining by Flow Cytometry C1qTNF5/CTRP5 Antibody (332923) [Unconjugated]](http://images.novusbio.com/fullsize/antibody/C1qTNF5_MAB3167_Flow_Cytometry_19754.jpg)
