Research of Por Deficiency has been linked to Congenital Adrenal Hyperplasia, Antley-bixler Syndrome, Autosomal Dominant, Hyperplasia, Adrenal Hyperplasia, Craniosynostosis. The study of Por Deficiency has been mentioned in research publications which can be found using our bioinformatics tool below. Researched pathways related to Por Deficiency include Excretion, Methylation, Pathogenesis, Localization. These pathways complement our catalog of research reagents for the study of Por Deficiency including antibodies and ELISA kits against 21-HYDROXYLASE, DSD, AR, BRCA2, CYP1A2.
Por Deficiency Bioinformatics Tool
Laverne is a handy bioinformatics tool to help facilitate scientific exploration of related genes, diseases and pathways based on co-citations. Explore more on Por Deficiency below!
For more information on how to use Laverne, please read the How to Guide.
We have 768 products for the study of Por Deficiency that can be applied to Western Blot, Chromatin Immunoprecipitation, Flow Cytometry, Immunocytochemistry/Immunofluorescence, Immunohistochemistry from our catalog of antibodies and ELISA kits.
Por Deficiency is also known as por deficiency, cytochrome p450 oxidoreductase deficiency, congenital adrenal hyperplasia due to apparent combined p450c17 and p450c21 deficiency, antley-bixler syndrome-like phenotype with disordered steroidogenesis, combined partial deficiency of 17-hydroxylase and 21-hydroxylase, antley-bixler syndrome with disordered steroidogenesis, congenital adrenal hyperplasia, antley-bixler syndrome, partialism, pord.